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DLBCL CAR-T Response Characterization

Characterize CAR-T response profiles by profiling tumor microenvironment before treatment

Identify T-Cell Exclusion Pre-Treatment

The Challenge

CAR-T Failure Rates Are High

CAR-T therapy achieves variable complete response rates in r/r DLBCL, but a substantial proportion of patients do not benefit. The high cost of manufacturing and treatment makes non-response a significant burden for patients and healthcare systems.

T-Cell Exclusion Is Associated with Failure

Patients with T-cell excluded tumors (fibrotic stroma, immune deserts) are associated with unfavorable CAR-T biology. CAR-T cells face barriers to tumor infiltration in these microenvironments. This is visible on H&E but not quantified pre-treatment.

No Pre-Treatment Biomarkers

Current practice: Treat all eligible r/r DLBCL patients with CAR-T, discover unfavorable outcomes post-treatment. Need informative biomarkers to characterize patient biological profiles BEFORE manufacturing CAR-T.

Wasted Manufacturing Costs

CAR-T manufacturing is costly per patient. Treating patients without favorable biological profiles wastes manufacturing capacity and delays access for patients with potentially favorable profiles.

"We treated a cohort of r/r DLBCL patients with CAR-T — a substantial fraction failed. Looking back at H&E slides, patients who did not benefit had obvious T-cell exclusion patterns. We should have characterized TME profiles before manufacturing."

The Helomnix Solution

Helomnix combines H&E image analysis with multi-omics profiling to characterize CAR-T response profiles BEFORE treatment. Here, cohort characterization focuses on describing tumor microenvironment states that influence CAR-T biology, rather than predicting individual patient outcomes. Our proprietary digital pathology models quantify tumor microenvironment features from pre-treatment H&E slides: T-cell infiltration, fibrotic stroma, immune exclusion zones, tumor-stroma architecture.

Multi-omics integration (RNA-seq, scRNA-seq) adds molecular context: immunosuppressive gene signatures, T-cell dysfunction markers, ABC vs. GCB subtype. Combined H&E + omics features enable comprehensive characterization of CAR-T response profiles.

This supports cohort enrichment: characterize which patients present favorable TME profiles (T-cell infiltrated tumors) versus unfavorable molecular context (biologically constrained for CAR-T activity), providing molecular context for translational and clinical research workflows.

Unique Differentiator

We integrate spatial H&E features (T-cell geography) with molecular features (gene expression) — neither alone is sufficient. Our paired H&E-RNAseq DLBCL training data captures this multimodal relationship.

How It Works

01

Pre-Treatment Data Collection

Obtain pre-treatment H&E slides (lymph node biopsy) and bulk RNA-seq or scRNA-seq from same sample. H&E captures spatial TME, omics captures molecular state.

02

H&E TME Profiling

Digital pathology AI quantifies: T-cell infiltration density, T-cell exclusion zones, fibrotic stroma area, tumor-immune interface length. Generates TME feature vector.

03

Multi-Omics Integration

RNA-seq profiling adds: immunosuppressive signatures, T-cell exhaustion markers, ABC/GCB classification. Integrates with H&E features.

04

CAR-T Response Characterization

Machine learning model trained on a curated DLBCL CAR-T cohort identifies molecular features associated with TME biology. Report: TME profile classification (favorable / intermediate / unfavorable molecular context) with key features driving characterization.

Real-World Application

DLBCL Use Case

A CAR-T treatment center sought to characterize patient profiles with favorable TME biology before initiating CAR-T manufacturing for r/r DLBCL.

Before

Standard approach: Treat all eligible r/r DLBCL patients (no biomarker selection). High non-response rate with significant wasted resources on patients who do not benefit.

After

Helomnix analyzed pre-treatment H&E and RNA-seq data from a retrospective cohort. Identified T-cell exclusion patterns and immunosuppressive gene signatures as features associated with unfavorable TME biology, enabling structured cohort characterization.

Outcome

Molecular context from TME characterization supported translational interpretation and informed study design considerations. The enriched cohort showed improved alignment between CAR-T biology and patient TME profiles.

Value to Your Organization

Improved

CAR-T Cohort Characterization

Improved characterization of patients with favorable CAR-T biology supports cohort enrichment and translational research workflows.

Substantial

Manufacturing Efficiency

Avoid manufacturing CAR-T for patients with unfavorable TME profiles, supporting meaningful reduction in wasted manufacturing costs.

Enhanced

Faster Patient Access

Better allocation of manufacturing capacity enables faster access for patients with favorable molecular profiles.

Our Methodology

Data Inputs

  • Pre-treatment H&E whole slide images (lymph node biopsy)
  • Bulk RNA-seq from same biopsy sample
  • ScRNA-seq (optional, for immune cell profiling)
  • Clinical metadata (stage, prior therapies, CAR-T product)
  • CAR-T outcome (CR, PR, NR, relapse, survival)

AI/ML Techniques

  • Automated H&E quality control
  • Digital pathology foundation models for feature extraction
  • Deep learning models for TME profiling from whole slide images
  • T-cell infiltration quantification from H&E
  • RNA-seq gene signature analysis (immunosuppressive and T-cell exhaustion programs)
  • ABC/GCB classification (COO subtype)
  • Multimodal fusion (H&E + omics) with ensemble learning
  • Feature attribution analysis for interpretability

Deliverables

  • CAR-T response characterization (structured risk assessment)
  • TME profile classification (favorable / intermediate / unfavorable molecular context)
  • TME profile: T-cell infiltration density, exclusion zones, fibrosis
  • Key informative features with importance scores
  • H&E attention heatmap highlighting influential regions
  • Gene signatures contributing to characterization
  • TME profile context to support translational interpretation for patients with unfavorable profiles
  • Summary report with molecular and TME characterization for translational review

Discuss a Translational Application

We welcome discussions about how this approach can support your translational research.